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Movie Title Year Distributor Notes Rev Formats 100 Creampie 2013 Soft on Demand Facial Squirt Creampie 100 Shots Drinker 2013 IdeaPocket Ai Uehara: Collector's Edition 2013 Soft on Demand All Night Drunk Creampie 2014 Moodyz Squirt Creampie Anal Creampie Fuck 2013 Soft on Demand Anal Debut Maid 2012 Moodyz Anal Doll 2012 Soft on Demand Anal DO Anal Fuck 2014 Moodyz Anal DO Anal Ravaged Young Wife 2013 Attackers AV Actress vs Her Brother 2013 Soft on Demand Bathing Time 2 2013 Soft on Demand Beautiful Hip Girl 2013 Soft on Demand Bukkake Creampie Anal Fuck: Ai Uehara 2014 Moodyz Anal Facial DP Swallow A2M Creampie Cohabitant Life with a Careless Sister 2014 Wanz Cosplay Fantasy 2014 BCDP Creampie at Share House 2014 HNDS Creampie Because I Love You 2013 Soft on Demand Creampie Creampie Cohabitant Life with Sisters 2014 Soft on Demand Facial Creampie Creampie Hot Spa 2013 Soft on Demand Creampie Double Paipan Splash 2012 Soft on Demand Double Real Creampie Special 2013 Moodyz CumSwap Creampie Drug Double Holes Acme Trip 2014 M's Video Group
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It is an essential mechanism involved in fluid balance.[1] It arises from a lack of fluids or an increase in the concentration of certain osmolites, such as sodium. If the water volume of the body falls below a certain threshold or the osmolite concentration becomes too high, structures in the brain detect changes in blood constituents and signal thirst.[2] Continuous dehydration can cause acute and chronic diseases, but is most often associated with renal and neurological disorders.[1] Excessive thirst, called polydipsia, along with excessive urination, known as polyuria, may be an indication of diabetes mellitus or diabetes insipidus. There are receptors and other systems in the body that detect a decreased volume or an increased osmolite concentration.[1][2] Some sources distinguish "extracellular thirst" from "intracellular thirst", where extracellular thirst is thirst generated by decreased volume and intracellular thirst is thirst generated by increased osmolite concentration.[2] Contents 1 Detection 1.1 Decreased volume 1.1.1 Renin-angiotensin system 1.1.2 Others 1.2 Cellular dehydration and osmoreceptor stimulation 1.3 Salt craving 1.4 Elderly 2 Thirst quenching 3 Neurophysiology 4 See also 5 References 6 Further reading Detection It is vital for organisms to be able to maintain their fluid levels in very narrow ranges. The goal is to keep the interstitial fluid, the fluid outside the cell, at the same concentration as the intracellular fluid, fluid inside the cell. This condition is called isotonic and occurs when the same level of solutes are present on either side of the cell membrane so that the net water movement is zero. If the interstitial fluid has a higher concentration of solutes than the intracellular fluid it will pull water out of the cell. This condition is called hypertonic and if enough water leaves the cell it will not be able to perform essential chemical functions. If the interstitial fluid becomes less concentrated the cell will fill with water as it tries to equalize the concentrations. This condition is called hypotonic and can be dangerous because it can cause the cell to swell and rupture. One set of receptors responsible for thirst detects the concentration of interstitial fluid. The other set of receptors detects blood volume.[2] Decreased volume Further information: Hypovolemia This is one of two types of thirst and is defined as thirst caused by loss of blood volume (hypovolemia) without depleting the intracellular fluid. This can be caused by blood loss, vomiting, and diarrhea. This loss of volume is problematic because if the total blood volume falls too low the heart cannot circulate blood effectively and the eventual result is hypovolemic shock. The vascular system responds by constricting blood vessels thereby creating a smaller volume for the blood to fill. This mechanical solution however has definite limits and usually must be supplemented with increased volume. The loss of blood volume is detected by cells in the kidneys and triggers thirst for both water and salt via the renin-angiotensin system.[2][3] Renin-angiotensin system Hypovolemia leads to activation of the renin angiotensin system (RAS) and is detected by cells in the kidney. When these cells detect decreased blood flow due to the low volume they secrete an enzyme called renin. Renin then enters the blood where it catalyzes a protein called angiotensinogen to angiotensin I. Angiotensin I is then almost immediately converted by an enzyme already present in the blood to the active form of the protein, angiotensin II. Angiotensin II then travels in the blood until it reaches the posterior pituitary gland and the adrenal cortex, where it causes a cascade effect of hormones that cause the kidneys to retain water and sodium, increasing blood pressure.[3] It is also responsible for the initiation of drinking behavior and salt appetite via the subfornical organ.[2] Renin-angiotensin-aldosterone system Others Arterial baroreceptors sense a decreased arterial pressure, and signals to the central nervous system in the area postrema and nucleus tractus solitarii.[2] Cardiopulmonary receptors sense a decreased blood volume, and signal to area postrema and nucleus tractus solitarii.[2] Cellular dehydration and osmoreceptor stimulation Main article: Osmoreceptor Osmometric thirst occurs when the solute concentration of the interstitial fluid increases. This increase draws water out of the cells, and they shrink in volume. The solute concentration of the interstitial fluid increases by high intake of sodium in diet or by the drop in volume of extracellular fluids (such as blood plasma and cerebrospinal fluid) due to loss of water through perspiration, respiration, urination and defecation. The increase in interstitial fluid solute concentration causes water to migrate from the cells of the body, through their membranes, to the extracellular compartment, by osmosis, thus causing cellular dehydration.[1] Clusters of cells (osmoreceptors) in the organum vasculosum of the lamina terminalis (OVLT) and subfornical organ (SFO), which lie outside of the blood brain barrier can detect the concentration of blood plasma and the presence of angiotensin II in the blood. They can then activate the median preoptic nucleus which initiates water seeking and ingestive behavior.[3] Destruction of this part of the hypothalamus in humans and other animals results in partial or total loss of desire to drink even with extremely high salt concentration in the extracellular fluids.[4][5] Imidazolium salt In addition, there are visceral osmoreceptors which project to the area postrema and nucleus tractus solitarii in the brain.[2] Salt craving


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